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Acute bilateral facial palsy and Wegener's disease.

Otolaryngol Head Neck Surg. 2008 Sep;139(3):476-7

Authors: Magliulo G, Parrotto D, Alla FR, Gagliardi S

PMID: 18722238 [PubMed - in process]

Monitoring vaccine safety using case series cumulative sum charts.

Vaccine. 2008 Aug 21;

Authors: Musonda P, Hocine MN, Andrews NJ, Tubert-Bitter P, Farrington CP

We adapt the self-controlled case series method for routine surveillance of vaccine safety using cumulative sum (CUSUM) charts. The CUSUM surveillance method we propose is applicable for detecting associations that arise in a short pre-determined risk period following vaccination. The performance of the case series CUSUM is investigated through simulations. We illustrate the method using retrospective analyses of influenza vaccine and Bell's palsy, and MMR vaccine and febrile convulsions.

PMID: 18723063 [PubMed - as supplied by publisher]

Treatment options for patients with Bell's palsy.

Am Fam Physician. 2008 Aug 1;78(3):316, 319; author reply 319-20

Authors: Sickels M

PMID: 18714457 [PubMed - in process]

Treatment options for patients with Bell's palsy.

Am Fam Physician. 2008 Aug 1;78(3):316; author reply 319-20

Authors: Irani J

PMID: 18711945 [PubMed - in process]

Congenital unilateral facial palsy and internal auditory canal stenosis.

Pediatr Neurol. 2008 Aug;39(2):116-9

Authors: Lin KM, Huang CC, Leung JH

Congenital stenosis of the internal auditory canal is a rare clinical entity. The majority of patients exhibit sensorineural hearing loss, and preserve facial-nerve function. We present a patient who manifested congenital unilateral facial palsy and ipsilateral sensorineural hearing loss clinically. Neuroimaging demonstrated stenosis of the unilateral internal auditory canal and hypoplasia of the ipsilateral trigeminal, facial, and vestibulocochlear nerves, despite normal function of the trigeminal nerves. The functional prognosis of facial and vestibulocochlear nerves was poor, and cognitive development demonstrated borderline delay.

PMID: 18639756 [PubMed - in process]

[Proportion of tau protein to phosphorylated tau protein CSF levels in differential diagnosis of dementia.]

Nervenarzt. 2008 Jul 16;

Authors: Hort J, Vališ M, Waberzinek G, Taláb R, Glossová L, Bojar M, Vyhnálek M, Skoda D, Masopust J, Sťourač P

BACKGROUND: The diagnosis of Creutzfeldt-Jakob disease (CJD) is based on typical clinical features and can be supported by detection of 14-3-3 protein in the CSF. The present study suggests the importance of investigating this ratio of total tau protein to phosphorylated tau protein in differentiating CJD from other dementias. Thirty-one patients with Alzheimer's disease (AD) or frontotemporal dementia and four with definitive diagnoses of CJD were included in the study. METHODS AND MATERIAL: Results from baseline investigations were compared with those from an age-matched cognitively controlled group with Bell's palsy. Tau protein, phosphorylated tau protein, and beta amyloid were analyzed using a commercially available enzyme-linked immunosorbent assay; 14-3-3 protein was assessed by Western blotting. RESULTS AND CONCLUSION: A distinctly high proportion of total tau protein to phosphorylated tau protein in CSF was found in all patients diagnosed with CJD, even in those with negative 14-3-3 protein blot results. In contrast, marker analysis in patients with Alzheimer's dementia revealed the highest CSF ratio of beta amyloid to phosphorylated tau protein levels. These proteins are important diagnostic biomarkers for CJD, especially in patients with negative 14-3-3 protein findings.

PMID: 18626620 [PubMed - as supplied by publisher]